Jul 22

Jul 22 The Mystery of SARS-CoV-2: Why does it kill some and not others?

The key to virus and vaccine side effects is the deadly spike protein that can bind to cells all over the body

PHOTO: Rutland (Mass.) Police Detective John Songy and his wife Joanne Songy. Both were hospitalized with Covid-19 in the spring of 2020. John didn’t make it.

By Tatiana Prophet

The SARS-CoV-2 virus attaches to a type of cell receptor that is found almost everywhere in the body, including white and red blood cells. So it would seem obvious why people have such widely differing symptoms. This is not just a respiratory disease.

The body’s ACE2 receptors are the locks that the virus’s iconic spike protein “key” penetrates. And these “locks” are found in the lungs, kidneys, gut and brain. This is why preventing cell entry is so important – and by many accounts, is not as complicated as we’ve been led to believe. Not all viruses are dependent on an acidic or neutral pH to achieve cell entry, but a review of the literature shows that SARS-CoV-2 does require an acidic environment (low pH).

“Since an acidic pH environment is favored for fusion and penetration of viruses into acidified endocytic vesicles (Gallagher et al. 1991), raising the extracellular pH is a potential method for blocking SARS-CoV-2 invasions,” wrote Bin Wang of Marshall University in the journal Genome.

Negatively charged ions and other ways of maintaining a higher pH have long been thought to maintain health and prevent disease. But recent social media posts with superficial errors have prompted fact checks stating “no, you can’t kill the coronavirus by eating an alkaline diet.”

Still, many left to guess what the next CDC guideline will be have been easing their fears with healthy food, clean air, exercise and an alkaline bodily environment.

The red blood cell attraction is particularly problematic but entirely preventable. If unchecked, the deadly spike protein can induce a rapid decline as the body is starved of oxygen. In one study published in Elsevier’s Emergency Collection in August 2020, Sean Hacking observed improvement with red blood cell transfusion for patients that were critical.

““Replenishing the oxygen carrying capacity of blood could be the key to addressing COVID-19 induced hypoxia and its downstream consequences,” wrote Sean M. Hacking in the journal Elsevier. “This is supported by a case report demonstrating a patient with cardiac arrest and multiple comorbidities including: chronic obstructive pulmonary disease, congestive heart failure, and anemia secondary gastrointestinal bleeding. The patient subsequently tested positive for SARS- CoV-2 and this ultimately progressed to pulmonary disease with bilateral interstitial infiltrates on his chest X-ray. The patient was anemic and treatment with packed red blood cells was undertaken. Initially, the patient was intubated for ventilation of acute respiratory failure. To the surprise of attending clinicians, oxygen stats improved and the patient was later extubated.””

— Sean Hacking, Elsevier Emergency Collection

And as early as May 2020, reports on CNN and National Geographic showed that ventilators being used on patients with hypoxia were not necessarily improving outcomes. Yet this news, obviously, went nowhere. And hundreds of thousands of patients were put on ventilators as they struggled to breathe from the Covid spike protein disabling their red blood cells. This was in spite of an article in the Boston Globe’s health news service, Stat News, stating that ventilators were being overused.

There are a lot of factors in determining just what this virus is and whether the measures taken were the most effective ones; but one key to understanding what Covid is (the flu, pneumonia, literally nothing, a lab accident or a bio weapon) is held within this entryway to so many cells.

It turns out that SARS1 (2003), SARS2 (2019), and a lesser-known HCoV-NL63, found in Amsterdam in 2004, are the only human coronaviruses known to use these abundant ACE2 receptors. But SARS2, according to multiple sources, binds much more tightly than SARS1. A deeper discussion from Back to Facts of the origins of both of these viruses can be found here.

Maybe that’s one difference and one reason why, outside of SARS1 AND SARS2, “most human CoVs … usually cause only mild respiratory diseases,” wrote Lobelia Samavati and Bruce D. Uhal. in the paper “ACE2: More than just a receptor for SARS-CoV-2.”

In fact, it was the receptor binding domain of SARS2 that drew attention to scientists speculating that perhaps the virus that causes Covid-19 had the hallmarks of genetic engineering.

But how to prevent this new pathogen from entering the cells? Surprisingly, there is quite a bit of scientific literature suggesting that the virus (and other viruses) require an acidic or neutral Ph environment to find a receptive cell.

Sometime in spring 2020, a Massachusetts police detective was hospitalized presenting with shortness of breath. He had tested positive for Covid-19 and told to go home and isolate — and seek help if things got worse. They got worse.

The man’s name was John Songy, and his entire community of Rutland was pulling for him. After his admission to the hospital, his condition deteriorated rapidly, and he was placed on a ventilator.

The man’s wife was hospitalized a few days later. Pictures of the couple showed them as relatively young, strong, and loving life. But it was the man who did not make it. And his wife, Joanne Songy, delivered his eulogy.

Songy was already suffering hypoxia when he came to the emergency room, which is a sign that Covid has entered the cells and is taking over the hemoglobin that delivers oxygen to the body. Joanne worked through social media to get him convalescent plasma, and he received it; but it was to no avail. Songy never came off the ventilator.

Why was it so hard for him to recover? Being slightly overweight, perhaps diabetic, having high blood pressure — we could all guess that it’s possible he had some of those conditions (but we don’t know).

But what we do know is that our society failed him because we failed to consider all the information available — even at that time — regarding cell entry.

When the fear and doubt creep in, we console ourselves that we do not have such metabolic preexisting conditions. But we wonder, why do some people inexplicably suffer the worst? And why do some with comorbidities recover? And why do some people experience lingering symptoms range from loss of taste and smell to chronic fatigue and brain fog?

More recently: And what do we do when the loved ones of departed Covid patients confront us to get the vaccine, and right away — not to question it.

These questions are important to everyone, and particularly those who want others to “not be selfish,” in other words to wear a mask and get vaccinated for the good of society. Yet how do we go take this experimental vaccine when we are not fully informed? That is what we aim to do here. If you have never heard of ACE2, or angiotensin or even spike proteins, then you are information-deficient.

According to the CDC, causality has not been established. However, most of these deaths occurred within a matter of days after getting the vaccine.

So far, nearly 11,000 people in the United States have died within days of getting the vaccine. As the CDC informs us, anyone can report to their “adverse event” vaccine website, VAERS.com. What they’re saying is that no causal relationship has been established. It doesn’t seem like they have any intention of establishing much, however. Another web site, openvaers.com, interprets the numbers from CDC site. I have personally checked these numbers and they are accurate.

Again, why people have such disparate outcomes from both the disease and the vaccine is held in this ACE2 receptor —. When ACE2 is compromised, if ACE2 is not in abundance, thrombosis and blood clotting can occur. And because the vaccines tell the body to create the deadly spike protein using genetic code, there is relatively little difference between the virus entering the cell, and the messenger RNA entering the cell: they’re both going to make the same spike protein that does not exist in the human body.

What’s to stop some people’s cells from creating spike proteins all over the body after receiving the vaccine? And what does the human body do with such a spike protein?

According to researchers in the UK, nothing. But they saw it as a good thing. “Professor Crispin said, "In this study we set out to see how closely the vaccine induced spikes resembled those of the infectious virus. We were really pleased to see a large amount of native-like spikes."

Here is what Nebraska Medicine had to say about the spike protein: “The spike protein is located on the outside of a coronavirus and is how SARS-CoV-2 (the coronavirus) enters human cells. Its location on the outside of the virus makes it so the immune system can recognize it easily.

“The spike protein is unique to SARS-CoV-2 – it doesn't look like other proteins your body makes. So antibodies created against the spike protein won't harm your body, they will only target coronavirus.”

Has there ever been harm from antibodies? Rather some of us need to be reassured that the spike proteins created in cells all over the body are not going to cause the very disease they’re aiming to prevent.

And: “Some have expressed concern that the spike protein or other parts of the mNA vaccines build up in the body, particularly in the ovaries or the brain. Here we break down the data to show where mRNA vaccines (and spike proteins) travel in the body. There is no evidence that any mRNA or protein accumulates in any organ.”

Yes, that’s what they said about DDT, aspartame, talcum powder, sunscreen and anti-smoking drug Chantix.

And why would a pause of the AstraZeneca vaccine, for example, be temporary if the evidence shows that the spike protein is causing blod clots? (AstraZeneca like the J&J, use a “harmless” adenovirus as a carrier for DNA that enters any cell with ACE-2 receptors and then integrates with that cell’s genetic code, and then that code creates the spike protein.)

Informed consent is the key. And we owe it to ourselves and our community to demand it.